- Warriors 2025 Offseason Tracker NBA
- Reports: Warriors add Jahmai Mashack in biggest NBA trade ever. Who is he? The Mercury News
- Warriors could block Tennessee star Jahmai Mashack’s move to Grizzlies in record-breaking NBA trade sportingnews.com
- VFL Jahmai Mayshack turns advice from father at age 12 into an NBA career Yahoo Sports
- Warriors’ Jahmai Mashack: Traded to Golden State CBS Sports
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Warriors 2025 Offseason Tracker – NBA
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This 100W Anker Block Charger Is Nearly Free, Amazon Is Selling Them by the Pallet Ahead of Prime Day
A built-in charger for a few extra port is a big plus, especially if you’re looking to declutter and charge several devices at once. Even better is that same charger being used with laptops, tablets, smartphones, and more. That’s exactly what the Anker Prime charger delivers, and right now it’s on all-time low during Amazon Prime Day. What makes this deal fantastic is that it’s open to everyone, not just Prime members, so there’s no subscription to pay to get the discount.
The Anker 100W 3-port Prime charger costs $49, down from its normal price of $69: This 29% price cut provides a fast and quality charging option at an affordable price if you need to charge multiple devices in a time-efficient way. With three ports (two USB-C and one USB-A), this charger can deliver up to 100W of total output, which makes it powerful enough to handle everything from a MacBook to the latest smartphones.
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100W 3-Port Block Charger
With this charger, you can fast charge multiple devices at the same time. Each USB-C port is capable of delivering a full 100W which means you can charge a 14-inch MacBook Pro from zero to 50% in just 30 minutes. This level of speed is a game-saver if you work from your laptop and need to top up quickly between meetings or flights. The third socket, a USB-A, provides you with even greater convenience so you can charge older devices or accessories that are still not on the USB-C bandwagon.
The thin design is also a huge plus: The charger is foldable prongs and it fits well into a bag or even a pocket and is perfect for travel or everyday use. Despite its tiny size, it’s built to last with thicker prongs for a better hold and a drop-proof design that can handle life on the go. The design is slim so it fits comfortably into the wall without shifting or falling out and you can count on it to keep on charging no matter where you leave it.
Anker Prime Charger is designed to work with a wide range of devices including MacBook Air and Pro, iPad Pro and Air, iPhone series, Galaxy S25 Series, Pixel phones, and even wearables like the Apple Watch and Galaxy Watch. Whether you’re charging a laptop, a phone or a tablet, you’ll have the right port and enough power to get the job done.
If you want to charge all of your devices with a single powerful charger, this Prime Day deal is one not to miss.
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New Meta AI Feature Raises Photo Privacy Concerns
Meta is asking users to grant its AI access to their unpublished photo libraries as it tests a new feature on Facebook, as first reported by TechCrunch. The company, which also owns Instagram, has admitted to using photos and text from public posts since 2007 to train its generative AI models, raising concerns about how Meta might use expanded access to personal information.
Some Facebook app users are encountering pop-ups asking them to grant Meta AI access to their camera rolls for “cloud processing.” The apparently new feature uses AI to “restyle” photos, group images by themes, like “birthdays or graduations,” and make “personalized creative ideas, like travel highlights and collages.” The notification says that Meta will periodically “select media” from users’ camera rolls based on time, location, and theme. It will not be used for “ad targeting,” it says.
In a statement to Hyperallergic, the company denied that it is using data from users’ photo libraries to train its AI models. However, Meta’s AI Terms of Service, to which users must accept in order to access the service, reserves the right to use “personal information” to “improve AIs and related technology.”
“By tapping ‘Allow,’ you agree to Meta’s AI Terms,” the feature’s pop-up states when users attempt to upload a Story on the app. “Media and facial features can be analyzed by Meta AI. To create ideas, we’ll use info like date and presence of people or objects.”
Facebook settings allow users to opt in or out of “camera roll cloud processing.” (screenshot Valentina Di Liscia/Hyperallergic) Meta introduced the function with three visual examples at the top, including an apparently enhanced photo of the Eiffel Tower, a trio wearing saturated clothes, and a digital collage titled “weekend recap.”
“We’re exploring ways to make content sharing easier for people on Facebook by testing suggestions of ready-to-share and curated content from a person’s camera roll,” a Meta spokesperson told Hyperallergic in a statement. “These suggestions are opt-in only and only shown to you — unless you decide to share them — and can be turned off at any time. Camera roll media may be used to improve these suggestions, but are not used to improve AI models in this test.”
The spokesperson did not provide an on-the-record response to questions about whether the new feature would be rolled out on its other platforms, when it was introduced, or if it would use data collected from unpublished images to train AI in the future
In the United States, Meta was not required to notify users that it was using public posts to train their AI, according to the New York Times. There are no opt-out options for US users, either. In Europe, however, stricter laws allow those on Instagram and Facebook to opt out of Meta’s data scraping project.
Artists have long raised concerns over the practice of training AI on publicly available images on the internet more generally, which they say allows the artificial intelligence models to replicate their artistic style. Some argue that the generative technology’s learned ability to mimic styles of artists could be detrimental to artists’ livelihoods.
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British Grand Prix: Lewis Hamilton says Ferrari was ‘most difficult car I’ve driven in these conditions’
Hamilton said Ferrari had upgrades coming to the car in forthcoming races – these would follow a new floor that was introduced at the previous race in Austria.
The 40-year-old said he wanted “a consistent balance, a car that turns at a low speed, just a more stable car”.
He added: “We were looking great through this weekend. In practice. we were always right up there. Still weren’t able to fully extract everything in qualifying. And then the race is a little bit harder. So that’s where I want to get stronger.”
Hamilton’s third fourth place of the season means he is still to finish on the podium since joining Ferrari at the start of this season.
Team-mate Charles Leclerc finished second-last in 14th after making the wrong call to come in for slick tyres on a damp track after the formation lap. His race spiralled downhill from there.
Leclerc said: “We were kind of nowhere the whole race, and when I say nowhere it’s like really nowhere, I was a second off (the pace) and on top of that I was doing lots of mistakes.
“I was really struggling to keep the car on track so it was an incredibly difficult day.”
Team principal Frederic Vasseur said: “The main issue we had today was that we struggled a lot when we were in the dirty air to overtake.
“We spent our lives overtaking sometimes 10 laps and then we were much faster. I think it was a difficult weekend, difficult for the strategy.”
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Patou Spring 2026 Ready-to-wear Runway, Fashion Show & Collection Review
“Joy always has a plan B,” said Patou’s artistic director Guillaume Henry, describing his fictional character of the season as an “outdoor woman” who is equally comfortable indoors.
That was a good thing given that the rain clouds looming over a post-heat wave Paris prompted a move of the show from the gardens of the Maison de la Chimie to the house’s oh-so-Parisian interiors, geometric parquet flooring, plaster mouldings and all.
As front row guest Susan Sarandon put it in a skit dropped on Instagram earlier in the day, “who the h–l is Joy?”
Despite sharing a name with the house’s historic bestselling scent, his fictional muse of the season doesn’t herald its imminent return, Henry said.
If the wardrobe the designer imagined is anything to go by, she’s a busy bee who doesn’t have time for outfit changes, expecting her sporty chic looks to work overtime from dawn to dusk.
The opening look, a straight black pinafore minidress with a pair of roomy patch pockets on the front, certainly fit the bill.
In pursuit of “fresh air, breathing room and lightness,” the designer imagined an encounter with house founder Jean Patou and his love of Art Deco lines and Christian Lacroix, who filled the house with feminine shapes and polka dots during his six-year tenure that began in 1981.
Cue a fusion with his proclivity for sporty knits, cropped proportions and utilitarian jackets that yielded long collarless styles with a gentle hourglass outline and versions with plunging trapeze necklines; shift dresses that could be dressed up or down with a canny change in accessories, and puffy skirts that came in anything from fine gray suiting to layered lace and exuberant peony prints — with pockets, of course.
A final trio gave a then-and-now vision of evening glamour, between floor-length bustier dresses and a lace bodysuit nodding to Sabrina Carpenter’s custom Patou look during her “Short n’ Sweet Tour.”
While continuing to build on the unfussy chic direction of the Henry era, the collection was in step with the dressier direction that emerged from June’s coed runways.
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Protein discovery could reverse Huntington’s disease damage, study finds
In the complex world of neuroscience, Huntington’s disease remains a cruel mystery. It slowly strips away a person’s ability to think and move, yet scientists still don’t fully understand why. Researchers at the University at Buffalo have spent over ten years trying to unravel that mystery. Now, they’ve uncovered two tiny but powerful proteins that may hold the key to stopping the disease before it causes irreversible damage.
What Happens in Huntington’s Disease?
Huntington’s disease is a rare but devastating condition caused by a genetic mutation in the huntingtin (HTT) gene. This mutation happens when the gene’s DNA code repeats a sequence—cytosine, adenine, guanine (CAG)—too many times. In people with Huntington’s, the number of repeats often goes beyond 36. That small change has big consequences: the altered gene creates a mutated HTT protein that leads to the death of brain cells.
Most people start to show symptoms around middle age. These can include trouble with movement, thinking, and mood. Eventually, the disease becomes fatal. For years, researchers knew that HTT helped with moving cell parts along thin structures called axons, which are like highways inside neurons. But no one could explain how the mutated version disrupted that process so badly.
The proteomic network on HTT containing light membranes is dramatically altered in HD iPSC-derived neurons. (CREDIT: Nature Cell Death & Disease) Cell Traffic and Dead Ends
In 2014, the same team at UB discovered that the normal HTT protein worked like a traffic controller. It helps move important cargo along the axons using tiny transport vehicles called vesicles. These vesicles travel thanks to motor proteins like kinesins and dyneins. Without HTT, that cargo gets stuck. Traffic jams form in the neurons, and cells begin to die.
That early discovery led to a bigger question: What tells HTT when to go, stop, or change direction?
This year, the team found the answer. Two signaling proteins, GSK3β and ERK1, help regulate that whole transport system. Both proteins are types of kinases, meaning they add small phosphate tags to other proteins to change how they function. But while both are involved, they have opposite effects.
The Good and the Bad
To test their theory, scientists used fruit flies genetically altered to have the same HTT mutation as in Huntington’s disease. When they blocked GSK3β, the flies showed fewer traffic jams in their neurons. Their cells were healthier, and the flies could even crawl better. But when they blocked ERK1, the opposite happened. More blockages formed, and more neurons died.
“With these findings, we propose that ERK1 may protect neurons in the face of Huntington’s disease, while GSK3β may exacerbate it,” said Dr. Shermali Gunawardena, a senior author on the study and associate professor at UB.
When the researchers increased ERK1 levels instead of blocking it, they saw reduced damage in the cells. That suggests that treatments boosting ERK1 or lowering GSK3β could one day help slow or even stop the disease.
“There’s not much that can be done once cells have died,” Gunawardena said. “So our whole research is trying to figure out these key, early processes that lead to cell death and whether that can be prevented.”
Pathogenic HTT triggers abnormal kinase associations with HTT and on membranes. (CREDIT: Nature Cell Death & Disease) Zooming in on the Cellular Map
To dig deeper, the team used stem cell-derived neurons from people with and without the HTT mutation. They isolated membrane structures from the cells and used advanced mass spectrometry to analyze the proteins attached to HTT. What they found shocked them: mutant HTT caused a dramatic shift in the types of proteins it interacted with.
In healthy neurons, HTT attached to proteins that support cell communication and transport. But in mutant cells, it was linked with different proteins—many of them involved in stress responses and cell death. This suggested that the mutated HTT protein wasn’t just broken. It was actively interfering with other essential cell processes.
They also saw higher levels of GSK3β and lower levels of ERK1 in the diseased neurons. Even more, the active form of GSK3β was significantly elevated, while the helpful form of AKT1, another regulator protein, was lower. That’s important because AKT1 normally keeps GSK3β in check. When AKT1 is down and GSK3β is up, it creates the perfect storm for neuron damage.
GSK3β and ERK phosphorylate non-pathogenic and pathogenic HTT in vitro. (CREDIT: Nature Cell Death & Disease) The fruit fly experiments confirmed these results. Blocking GSK3β in flies reduced both transport blockages and brain cell death. On the other hand, reducing ERK1 levels made things worse—more damage, more dysfunction. But when ERK1 was boosted, many of those problems improved.
“The level of ERK1 is clearly important for Huntington’s disease,” said Thomas J. Krzystek, the study’s first author. “Even if we don’t know exactly how it works, the pathway clearly protects neurons.”
A Path to Better Treatments
Scientists now believe that the early stages of Huntington’s disease involve a disruption of HTT’s normal role as a scaffold. In healthy cells, HTT helps bring other proteins together at membranes, like parts on a workbench. These include the motor proteins that move cargo and the membrane proteins that receive signals from the rest of the body. But when HTT is mutated, it can no longer hold these pieces in place.
As a result, entire signaling networks break down. The kinases that should be moving around the cell end up clumping together or disappearing from key areas. That makes it harder for neurons to maintain communication and health.
GSK3 inhibition mitigates larval locomotion defects, axonal transport blockages, abnormal synaptic morphology, and elevated neuronal cell death elicited by pathogenic HTT. (CREDIT: Nature Cell Death & Disease) Among the biggest disruptions was in a group of pathways related to axon guidance, membrane trafficking, and vesicle transport. These pathways are vital for neuron survival. In the diseased cells, proteins like RAB7 and kinesin-1 showed abnormal patterns. That suggests they were either being trapped by the mutant HTT or unable to reach their proper locations.
These findings are significant because both GSK3β and ERK1 are already targets in other areas of drug development. Small molecule inhibitors for GSK3β and activators for ERK1 exist and are being explored in diseases like Alzheimer’s and cancer.
“Future treatment could potentially increase a patient’s levels of ERK1 to mitigate their neuronal cell death,” Gunawardena said. “That would need to be done carefully so it doesn’t affect other processes.”
ERK does not play a major role in axonal transport regulation, unlike GSK3β. (CREDIT: Nature Cell Death & Disease) Published in the journal, Nature Cell Death & Disease, the work was funded by the National Institute of Neurological Disorders and Stroke, as well as support from UB’s Mark Diamond Research Fund, the Stephanie Niciszewska Mucha Fund, and the BrightFocus Foundation.
Though Huntington’s disease has no cure today, the research brings new hope. By targeting key regulators like GSK3β and ERK1, scientists are getting closer to slowing or stopping the condition before damage becomes permanent. And with each new discovery, the tangled web of HTT and its deadly mutation becomes a little clearer.
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‘Animal Shelter 2’ Heads for PC 23 July
Developer Games Incubator announced that their pet pound simulator ‘Animal Shelter 2’ will launch on PC via Steam on the 23rd of July.
Welcome back to the heartwarming world of Animal Shelter 2! Provide a loving home for every four-legged friend in need – play with them, pet them, even dress them up! Get to know them and find them a loving family. Play with your friends and manage your shelter together in co-op!
Have a gander below for more details on Animal Shelter 2, including details on the last fundraiser the development team put together for animals in need.
Games Incubator has officially announced that Animal Shelter 2 will launch on PC via Steam on July 23. Time to visit the coziest shelter in town – and this time, you don’t have to handle the responsibility alone. Thanks to new online co-op mode, players can team up with friends to build a top-notch haven for their furry friends!
Animal Shelter 2 isn’t just a bigger version of the original – it’s packed with a range of new features. The sequel brings visual upgrades for both animals and the surroundings, and now your shelter is also fully customizable! Give it a name (visible in-game), resize rooms, repaint walls, or add decorations – some of which will directly improve the comfort of your animals, your staff, and visitors, increasing the chances for adoption!
You’ll also be able to adopt a pet yourself, as well as support donations or other forms of aid available on the map – in addition to the main area, there will be interventions in three types of procedurally generated locations, making each new spot even more unique!
But that’s just the beginning! Here’s what else awaits future caretakers in Animal Shelter 2:
🤝New Mode – Co-op: Taking care of animals is serious business – but it’s a lot easier (and way more fun) with friends. In online co-op mode, you and your dream-team can manage the same shelter together – share the workload or wipe each other’s tears, the choice is yours.🦸Staff & Volunteers System: Hire NPCs in special uniforms and welcome volunteers to your team. Build a perfect crew dedicated to giving animals the care they deserve.
💊Advanced Medical System: Sometimes a quick help may not be enough – chronic conditions require you to monitor medicine doses closely, and in complicated cases, your furry patient may need external testing. Once the health issues are under control, you ‘just’ need to tackle those tricky behavioral issues!
🍂Day/Night & Seasonal Cycles: Tired after the night shift? Well, that’s part of the job! Keep your shelter cool in the summer and warm in the winter. Don’t forget to shovel snow, patch up leaky roofs, and keep your animals dry and cosy – when wet or cold, pets are more likely to get seriously sick.
Thanks to the enthusiastic player support, the team is also continuing the community-nominated shelter fundraisers, which have previously helped deliver over $30,000 to animals in need. Submissions for this new round will open soon via Steam and a dedicated form. Learn more about the last fundraiser HERE.
Animal Shelter 2 launches July 23, 2025, on PC via Steam. Add it to your wishlist today, and don’t miss a single bark! For more details, check out the official Steam page.
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‘Zenless Zone Zero’ Version 2.1 “The Impending Crash of Waves” Lands 16 July
Last Friday, publisher HoYoverse announced that their free-to-play urban fantasy action title ‘Zenless Zone Zero’ will see its next update, Version 2.1, land across all platforms on the 16th of July. Dubbed “The Impending Crash of Waves”, summer-themed update introduces Season 2 Chapter 2 and introduces the Spook Shack faction alongside two new regions. Free outfits, two new agents and a Miyabi rerun, events, and more await players in ZZZ’s latest version update.
Have a gander below for more details on Zenless Zone Zero’s 2.1 update. The game is available on PC via HoYoPlay, PlayStation 5, Xbox Series consoles, iOS, and Android via Google Play—with Xbox Game Pass members seeing monthly bonus drops like those found in ‘Genshin Impact’.
In Version 2.1, Proxies can explore two exciting new additions: Sailume Bay and Fantasy Resort. Nestled beneath the cliffs of Failume Heights, Sailume Bay will become the central stage for Chapter 2’s story. Here, Porcelumex will launch a comprehensive safety investigation to resolve conflicts stemming from the chaos caused by the Exaltists.
The high-profile investigation team includes Alice Thymefield, descendant of the esteemed Thymefield family, who will later join the Spook Shack. Meanwhile, the summer story unfolds at Fantasy Resort, featuring sandy beaches, coconut trees swaying in the breeze, and a vibrant holiday atmosphere perfect for summer activities and mysterious stories.
Ukinami Yuzuha 
Alice Thymefield Two remarkable allies from the Spook Shack will join the roster. Ukinami Yuzuha is a shrewd S-Rank Physical Support Agent who deploys her tanuki companion Kama to throw candy bombs at enemies, empowering on-field attackers to accumulate Anomaly Buildup more easily. Her signature Sweet Scare ability adapts to the active agent’s attribute via Flavor Match, significantly boosting the Anomaly Buildup efficiency while simultaneously triggering Aftershock attacks.
Alice Thymefield, the newest member of Spook Shack, is an S-Rank Physical Anomaly Agent. As a noble heiress, she utilizes her refined swordsmanship techniques to maneuver on the battlefield and accumulate Blade Etiquette. By consuming Blade Etiquette, Alice can unleash powerful strikes, amplifying Assault and Disorder damage. Meanwhile, Miss Esme, a graceful S-Rank Bangboo, soars alongside agents while generating beneficial effects that replenish Energy or HP. Additionally, the Void Hunter Miyabi (S-Rank Frost Anomaly) and Yanagi (S-Rank Electric Anomaly) will rerun on the Version 2.1 banners.
The once-pristine Fantasy Resort now stands as a fading paradise awaiting restoration, and Proxies will partner with the Spook Shack to revive the resort through a series of aquatic activities and attractions. Proxies can enjoy the thrills of surfing by navigating obstacles with power-ups, or board yachts and embark on exciting sea shooting challenges amidst scenic island tours.
New fishing spots await our experienced anglers, featuring fresh new species and optimized interactions with reduced grinding for a more relaxed yet diverse gameplay. Besides the summer activities, stylish outfits that fit the summer vibes will be available for Yuzuha, Alice, and the protagonists. In addition, a new Bangboo Auto Battler event, “En-Nah” Chess Legend, will be introduced with a PVP mode, and the Simulated Battle Trial will be refreshed with a new scoring system that rewards combat technique mastery for challenging combat enthusiasts.
Zenless Zone Zero Version 2.1 “The Impending Crash of Waves” launches on July 16 with optimizations to provide a smoother summer adventure. To show our appreciation for your continued support, Proxies can claim 1,600 Polychromes before Version 2.1 ends. Rated Teen by ESRB and PEGI 12, the game is available on PlayStation®5 Pro, PlayStation®5, Xbox, PC, iOS, and Android. For more information, visit https://zenless.hoyoverse.com/ or follow @ZZZ_EN on X.
ンナァー! (How graceful!) Continue Reading
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Ford CEO sounds alarm on China’s EV dominance — what that means for you
Ford CEO Jim Farley didn’t mince words at the recent Aspen Ideas Festival, describing China’s rapid rise in the electric vehicle (EV) market as the “most humbling experience” of his career.
“Their cost, their quality of their vehicles is far superior to what I see in the west.” Farley said.
Chinese automakers like BYD have pulled ahead with vertically integrated supply chains, efficient production and government support. They’re pumping out reliable EVs at prices that make even budget-conscious U.S. models look expensive — including the new BYD Seagull, priced under $10,000 USD.
“We are in a global competition with China, and it’s not just EVs. And if we lose this, we do not have a future Ford,” Farley said.
For U.S. consumers, this isn’t just about bragging rights in the global auto race. It’s about what you’ll pay for your next vehicle.
If Chinese EVs enter Western markets without tariffs, they could undercut competitors by tens of thousands of dollars — bringing huge savings to consumers but serious threats to U.S. auto industry jobs. In response, the U.S. government has already imposed steep tariffs on Chinese electric vehicles, which may shield domestic automakers for now but also delay price competition.
Farley’s warning comes as more and more Americans are looking to switch to EVs amid soaring gas prices and expanding charging infrastructure. But since Tesla, Ford and GM EVs often start in the $40,000-$60,000 range, many Americans are priced out.
Read more: No millions? No problem. With as little as $10, here’s how you can access this $1B private real estate fund of diversified assets usually only available to major players
China’s advantage? It controls much of the world’s battery production and can bring new EVs to market in a fraction of the time compared to U.S. automakers.
Ford is working on a next-gen affordable EV platform intended to match China’s costs but says it won’t arrive until 2027. Tesla is also aiming to launch a $25,000 “Model 2,” but timelines remain uncertain. Until then, the price gap persists.
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Lewis Hamilton admits his Ferrari was ‘very hard to drive’ at Silverstone after P4 finish
Lewis Hamilton assessed the British Grand Prix as a “difficult race”, with the seven-time World Champion admitting that his Ferrari had been a “very hard car to drive” in the mixed conditions at Silverstone.
After showing flashes of strong pace during the weekend, Hamilton lined up in P5 on the grid for his home race and suggested that he would not give up hope of a podium.
That dream very nearly came true as the 40-year-old found himself in fourth and chasing down third-placed Nico Hulkenberg in the latter stages of the Grand Prix, before ultimately missing out on the rostrum by just over five seconds.
Sharing his reflections after jumping out of the car, Hamilton said: “[It was a] difficult race, not a great one from my side. I did the best I could.
“As it showed there was plenty of mistakes out there and very, very tricky conditions, in really a very, very hard car to drive here – this car doesn’t like those conditions. Not ideal, but we got some points at least.”
Hamilton’s pace appeared to drop off slightly as he embarked on a second stint on the intermediate tyres before switching to the soft compound in the final phase.
Asked to summarise that segment of the race, he explained: “The tyres [were] dropping off, the car doesn’t like low-speed corners, lots of understeer, and I started knocking out the left front and then just couldn’t keep up.”
Despite the disappointment of missing out on a top-three result, Hamilton could still take the positives from racing in front of his home crowd.
“What I take from it is the amazing energy from all the fans,” the Briton said.
“They’ve been incredible this weekend and it’s been amazing to see so many people switch to red and, yeah, got some points.”
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