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  • Spotify-like AI helps discover never-before-seen supernova as greedy star attempts to eat a black hole

    Spotify-like AI helps discover never-before-seen supernova as greedy star attempts to eat a black hole

    Scientists may have spotted a never-before-seen kind of supernova, after using a Spotify-like artificial intelligence (AI) to scan the skies for strange activity.

    The AI unearthed signs of what could have been a huge star blowing up just as it was attempting to gulp down a nearby black hole.

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  • TRAPPIST-1d isn’t the Earth-like planet scientists had hoped it to be, according to JWST data

    TRAPPIST-1d isn’t the Earth-like planet scientists had hoped it to be, according to JWST data

    There’s bad news for our hopes of habitable planets existing around TRAPPIST-1, with the James Webb Space Telescope (JWST) finding no evidence for an Earth-like atmosphere on a third world orbiting the red dwarf.

    However, that still leaves four other planets in orbit around TRAPPIST-1 that could be habitable, with at least two or three of them in what is regarded as the “habitable zone” where temperatures would be suitable for liquid water to exist —- assuming an Earth-like atmosphere that can retain heat.

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  • Incredible turnaround lifts New Zealand past Lebanon into Semi-Finals

    Incredible turnaround lifts New Zealand past Lebanon into Semi-Finals

    JEDDAH (Saudi Arabia) – New Zealand staged the biggest comeback of the FIBA Asia Cup 2025 so far, erasing a 22-point deficit to shock Lebanon 90-86 in Thursday’s Quarter-Finals at King Abdullah Sports City.

    The Tall Blacks looked dead in the water after trailing 35-13 early in the second quarter, but Mojave King and Max Darling spearheaded a relentless fightback that sent the 4-0 Group D winners into the Semi-Finals.

    Darling finished with 18 points, 9 rebounds, 4 assists and a block, battling inside to keep the Kiwis afloat and delivering timely scores in the rally. King was electric with 23 points, 3 assists, 2 triples and 2 steals, igniting the decisive fourth-quarter push. Flynn Cameron added 12 points and 3 assists while Tohi Smith-Milner and Carlin Davison came up big down the stretch with clutch plays at both ends.

    For Lebanon, Dedric Lawson was a force with 24 points, 13 rebounds and 2 assists, while Hayk Gyokchyan buried five triples en route to 21 points. Ali Mansour and Amir Saoud combined for 24 points but could not stem the late surge.

    “I think we deserved to win, but again we look at the mirror and each of us made mistakes, especially me,” Saoud said. “I should have done a better job leading the team.”

    Lebanon’s opening quarter was nothing short of explosive. Karim Zeinoun and Gyokchyan hit five triples between them, Khayat and Lawson ran the break to perfection and their swarming defense left New Zealand searching for answers. The 20-point cushion seemed safe, especially as Lebanon still led 45-30 at halftime.

    The game flipped in the third. New Zealand attacked the paint with purpose, found rhythm from deep and cut the gap to just six heading into the final quarter. The momentum carried over as Smith-Milner’s outside shooting and Taylor Britt’s drives chipped away at the lead before King tied it at 72-all with under four minutes remaining. Moments later, his fastbreak three-point play gave the Tall Blacks their first lead of the game.

    Lebanon refused to fold, with Gyokchyan’s clutch triple and Mansour’s timely scores keeping it close. However, Davison’s emphatic dunks, Taine Murray’s cold-blooded three and Cameron’s late putback sealed an incredible come-from-behind victory for the Kiwis.

    “It was a ballgame. Lebanon, in that first quarter, could do no wrong. They made us pay for every mistake that we had,” New Zealand coach Judd Flavell said. “We were in a big hole, we had to make some adjustments and we did. The mindset was get to the rim, and we were able to turn it around. So proud of this young group to grind it out in the end. It was also probably Max Darling’s best game in a Tall Blacks singlet.”

    The result sends New Zealand to the Semi-Finals, where they will face China for a place in the gold medal game. For Lebanon, the loss ends their title aspirations, but they go home with heads held high after a stirring campaign in Jeddah.

    From staring down elimination to celebrating one of the tournament’s most remarkable turnarounds, New Zealand have shown they are not just unbeaten, they are unbreakable.

    FIBA

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  • Linking Planet Formation To Exoplanet Characteristics: C/O As A Diagnostic Of Planet Formation

    Linking Planet Formation To Exoplanet Characteristics: C/O As A Diagnostic Of Planet Formation

    Global atmospheric C/O values for directly-imaged (blue) and transiting (purple) planets as a function of semi-major axis (left) and planet mass (right). This plot is reproduced using the data compiled in Hoch et al. (2023). Added to this plot are values from current C/O values reported in the literature (Ahrer et al. 2023; Alderson et al. 2023; Feinstein et al. 2023; Rustamkulov et al. 2023; Tsai et al. 2023; Edwards & Changeat 2024; Smith et al. 2024; Xue et al. 2024; Bell et al. 2023; Radica et al. 2023; Taylor et al. 2023; Coulombe et al. 2023) from the analysis (or inclusion) of data from JWST (orange). Error bars are included only if reported. The dotted horizontal line shows the solar C/O value (0.58; Asplund et al. 2021). — astro-ph.EP

    Gas-giant exoplanets are test cases for theories of planet formation as their atmospheres are proposed to carry signatures of their formation within the protoplanetary disk.

    The metallicity and C/O are key diagnostics, allowing to distinguish formation location within the disk (e.g., relative to snowlines), and mechanism (e.g., core accretion versus gravitational instability).

    We can now probe the composition of the planet-forming regions of disks, and that in gas-giant exoplanets, to scrutinise these theories and diagnostics. So far, ALMA has revealed that the outer disk regions are typically metal-depleted and O-poor, whereas JWST is showing that the inner disk regions around Sun-like stars are mostly O-rich.

    Further, JWST is showing that most transiting gas-giant planets are typically metal-enriched and O-rich, consistent with formation at/within the water snowline and pollution by icy bodies. There is emerging an arguably “conventional” picture of gas-giant planet formation for transiting planets, to be confirmed, of course, with future data.

    Catherine Walsh (University of Leeds)

    Comments: Accepted for publication in IAU Symposium 383 proceedings “Astrochemistry VIII: From the first galaxies to the formation of habitable worlds”. This short review was composed in January 2024 and all references/information are up to date until that time
    Subjects: Earth and Planetary Astrophysics (astro-ph.EP); Astrophysics of Galaxies (astro-ph.GA)
    Cite as: arXiv:2508.09587 [astro-ph.EP] (or arXiv:2508.09587v1 [astro-ph.EP] for this version)
    https://doi.org/10.48550/arXiv.2508.09587
    Focus to learn more
    Submission history
    From: Catherine Walsh
    [v1] Wed, 13 Aug 2025 08:02:48 UTC (656 KB)
    https://arxiv.org/abs/2508.09587
    Astrobiology,

    Explorers Club Fellow, ex-NASA Space Station Payload manager/space biologist, Away Teams, Journalist, Lapsed climber, Synaesthete, Na’Vi-Jedi-Freman-Buddhist-mix, ASL, Devon Island and Everest Base Camp veteran, (he/him) 🖖🏻

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  • El Salvador Greenlights Investment Banks, Marking a New Chapter in Global Finance – Dentons

    1. El Salvador Greenlights Investment Banks, Marking a New Chapter in Global Finance  Dentons
    2. El Salvador passes law letting investment banks hold bitcoin, serve qualified clients  The Block
    3. El Salvador’s Bitcoin (BTC) Holdings Top $750 Million: Trading Alert and Verification Status  Blockchain News
    4. Will Bitcoin banks in El Salvador trigger a new crypto bull run—and which altcoins could 10x from it  AInvest
    5. El Salvador hints at Bitcoin banks as $400 trillion wealth shift gains steam  CryptoSlate

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  • Smartphone app doubles quit rates among low-income smokers

    Smartphone app doubles quit rates among low-income smokers

    While smoking rates in the United States have substantially declined over the past six decades, smoking remains high among people with low incomes, leading to health disparities. A smartphone app that delivers real-time, tailored messages may hold the key to helping them quit, according to University of Oklahoma clinical trial results published today in JAMA Network Open.

    The clinical trial compared two different smartphone tobacco cessation apps: Smart-T, which was developed by OU researchers, and QuitGuide, a product of the National Cancer Institute. Smart-T is designed to gauge users’ risk for smoking throughout the day and to respond in real time with custom messages depending on what people are experiencing. QuitGuide is a static app that allows users to track their cravings and provides tips for resisting the urge to smoke. After six months of using the apps, those who used Smart-T were nearly twice as likely to have quit smoking than those who used the QuitGuide app.

    “Smart-T is like having a tobacco cessation counselor in your pocket. While a combination of behavioral counseling and medication is the most effective way to quit smoking, in-person counseling may not be practical for everyone, especially those facing barriers like transportation or busy schedules. With Smart-T, our motivation is to make the app comparable to standard counseling treatments,” said the study’s lead author, Emily Hébert, DrPH, a member of the TSET Health Promotion Research Center (HPRC) at OU Health Stephenson Cancer Center and an assistant professor of family and preventive medicine in the OU College of Medicine.

    During the trial, which enrolled 454 low-income smokers from across the United States, half were randomly assigned to use Smart-T and the other half QuitGuide. Those using Smart-T received up to five prompts a day to provide input about their urges to smoke, whether they were around other smokers, their stress level, their mood and other variables that are known to increase the risk of smoking. Based on their answers, the app’s algorithm created a risk score and then responded with messages tailored to each person’s particular type of risk, such as a suggestion to do a breathing exercise or to use nicotine replacement therapy. Participants in both groups were provided nicotine patches or gum or lozenges.

    Not only were Smart-T users significantly more likely to quit smoking after six months compared to those using the QuitGuide app, we also found that they used Smart-T more, found it more helpful, and were more likely to ask for more nicotine therapy if they ran out.”


    Emily Hébert, DrPH, member of the TSET Health Promotion Research Center (HPRC), OU Health Stephenson Cancer Center and assistant professor of family and preventive medicine, OU College of Medicine

    Participants’ smoking status was verified when they blew into a device connected to the phone, which measures carbon monoxide in their breath. Facial recognition software confirmed the participant was the one using the device. Hébert said this is one of the first trials testing a mobile app to verify participants’ smoking status biochemically.

    Next steps are to study Smart-T in larger groups across the United States and with follow-up times longer than six months. “Some people who smoke will not benefit from apps like Smart-T, but low-cost and always-available smartphone interventions could provide a convenient way for low-income adults to quit. We’re really trying to find the best recipe for smoking cessation for everyone,” she said.

    About the project

    The publication, “Just-in-Time Adaptive Intervention for Smoking Cessation in Low-Income Adults,” can be found at https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2837640.

    The Smart-T app was created by Michael Businelle, Ph.D., who is co-leader of the next research phase with Hébert. With Hébert as assistant director, Businelle leads the mHealth Shared Resource, which launched the Insight™ mHealth Platform in 2015 to create and test technology-based interventions. The efforts are part of the TSET Health Promotion Research Center (HPRC), a global leader in mobile health technology that has supported more than 115 studies and attracted $85 million in grants. Businelle is co-director of HPRC and is a professor in the Department of Family and Preventive Medicine in the OU College of Medicine.

    The research described in this news release was supported by the National Cancer Institute (grant No. R01CA221819) and used the mHealth Shared Resource, which is partially funded by the NCI (grant No. P30CA225520). Additional support was provided by the Oklahoma Tobacco Settlement Endowment Trust and the Oklahoma Shared Clinical and Translational Resources through an Institutional Development Award from the National Institute of General Medical Sciences (grant No. U54GM104938).

    Source:

    Journal reference:

    Hébert, E. T., et al. (2025). Just-in-Time Adaptive Intervention for Smoking Cessation in Low-Income Adults: A Randomized Clinical Trial. JAMA Network Open. doi.org/10.1001/jamanetworkopen.2025.26691.

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  • Research sheds new light on ALS gene mutation

    Research sheds new light on ALS gene mutation

    Researchers at Yale School of Medicine said they’ve solved a longstanding mystery of how mutations in the C9ORF72 gene, a common genetic cause of amyotrophic lateral sclerosis (ALS), may lead to toxic proteins.

    The findings suggest a new approach that could lead to the development of targeted therapies, the researchers said.

    Their study, “Aberrant splicing exonizes C9orf72 repeat expansion in ALS/FTD,” was published in Nature Neuroscience.

    The discovery centers on mutations in C9ORF72, which are the most common genetic cause of ALS in European and North American populations. Although the causes of ALS aren’t fully understood, genetic mutations are known to be behind at least some cases of the disease.

    Within the C9ORF72 gene, there’s a region where a specific genetic sequence is normally repeated about a dozen times, but in ALS-causing mutations, the sequence is repeated hundreds of times. Researchers have known that this expanded genetic repeat leads to the production of toxic proteins that are thought to drive nerve cell damage in ALS.

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    ALS gene mutation contains a splicing puzzle

    Genes are made of exons — the sections that provide instructions to make proteins — and introns, which are regions that don’t code for proteins but help regulate gene activity. Within a cell’s DNA, genes contain exons interspersed with introns.

    When the gene is read to make protein, the entire gene is copied over into a temporary molecule called messenger RNA (mRNA). The mRNA then undergoes a process called splicing, where the introns are removed and the exons are strung together to form a sequence that is ultimately used to make proteins.

    The ALS-causing expansion mutation in C9ORF72 is located in an intron, which should normally be removed by splicing before the gene’s mRNA is used to make any protein. And yet, it’s been established that the mutation does lead to the production of abnormal proteins.

    Although these mechanisms are well established, they’ve led to a conundrum for researchers: How could a mutation in an intron, a part of the gene that should be discarded, lead to a toxic protein?

    “These repeat proteins can interfere with a wide range of cell functions,” Suzhou Yang, a PhD student in Yale’s interdepartmental neuroscience program and lead author of the study, said in a university news story. “But it has been a mystery how an intronic sequence, which is usually cut out and degraded, can be translated into these toxic proteins.”

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    An illustration showcasing a person's brain.

    Mystery solved

    The scientists figured out an answer to this puzzle. They determined that ALS-causing mutations in the C9ORF72 gene cause problems with the molecular machinery that normally controls mRNA splicing. As a result, part of an intron — including the mutation — instead turns into an exon and is included in the mature mRNA used to make protein.

    Junjie Guo, PhD, associate professor of neuroscience at Yale and senior author of the study, said this shows that “we must be cautious about categorizing mutations simply according to existing gene models, because it turns out, an intron does not always stay an intron.”

    “Part of the reason why it took us so long to find this is perhaps our static way of thinking about the genome and gene expression,” Guo said. “But once we saw the RNA sequence, this abnormal process immediately jumped out to us.”

    The discovery opens potential avenues for therapy, the researchers said. For starters, they showed that they could reduce expression of the mutant mRNA by using a small piece of genetic material to target a sequence that’s only present when the intron is included.

    This allowed the treatment to target only the faulty mRNA without affecting the normal one, enabling cells to keep producing the healthy version of the protein.

    “From the therapeutic perspective, we believe that this is a broadly applicable strategy of identifying unique sequences that could allow us to design therapeutic candidates to selectively target the disease-causing RNA,” Guo said.

    The scientists hope to learn more about how mutations disrupt splicing to change an intron into an exon. Once those mechanisms are more fully understood, it may be possible to target them therapeutically.

    “It is most likely that there are additional cell-type-specific splicing regulators that determine the incorrect splice sites,” Guo said. “If we can identify these regulators, they could become potential targets for manipulating and reversing these abnormal events.”

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  • Decoding Mohs Micrographic Surgery Through an Artificial Intelligence-Powered Search Analytics Approach

    Decoding Mohs Micrographic Surgery Through an Artificial Intelligence-Powered Search Analytics Approach


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  • Trump says he thinks Putin will ‘make a deal’ on Ukraine | Russia-Ukraine war News

    Trump says he thinks Putin will ‘make a deal’ on Ukraine | Russia-Ukraine war News

    The US president’s words come on the eve of his closely watched Alaska summit with Russian President Vladimir Putin.

    One day before the leaders of Russia and the United States are set to meet in the US state of Alaska to discuss ending the Russia-Ukraine war, US President Donald Trump said he believes his Russian counterpart is ready “to make a deal”.

    In an interview on Fox News Radio on Thursday, Trump said he believed Russian President Vladimir Putin “wanted the whole thing” – in an apparent reference to his territorial aspirations in Ukraine – but was willing to come to the table and make a deal due to the relationship between the two men.

    “I think he wants to get it done. I really feel he wanted the whole thing. I think if it weren’t me, if it were somebody else, he would not be talking to anybody,” Trump told interviewer Brian Kilmeade.

    Trump and Putin will meet in Alaska on Friday for talks on the more than three-year conflict. Ukrainian President Volodymyr Zelenskyy will not be present at the talks, though Trump has said that should Putin signal a willingness to end the war, another meeting between the two leaders would follow.

    “I don’t know that we’re going to get an immediate ceasefire, but I think it’s going to come. See, I’m more interested in an immediate peace deal – getting peace fast. And depending on what happens with my meeting, I’m going to be calling up President Zelenskyy and [saying] let’s get him over to wherever we’re going to meet,” Trump said.

    He added that there was the possibility they could simply “stay in Alaska”, but also stressed that if the meeting went poorly, “I’m not calling anyone. I’m going home.”

    That hedging represents a seeming cautiousness by Trump, who has spoken about being frustrated by Putin’s broken promises in the past.

    Speaking from Washington, DC, Al Jazeera’s Shihab Rattansi said Trump’s metric for success could be boiled down to what read he had on Putin.

    “He very much made it clear that what success means in this context is him being convinced that Vladimir Putin is serious about peace, and then arranging a second meeting that would involve the Ukrainians,” he said.

    Earlier on Thursday, Putin praised Trump, saying he was “making quite energetic and sincere efforts to end the fighting”.

    The words came shortly after Zelenskyy met with British Prime Minister Keir Starmer in London, where they discussed security guarantees for Ukraine that could “make peace truly durable if the United States succeeds in pressing Russia to stop the killings and engage in genuine, substantive diplomacy”, Zelenskyy wrote on X.

    The meeting, said Al Jazeera’s Jonah Hull, “was about a show of unity ahead of that summit in Alaska”.

    Hull noted there was a “sense of some optimism” following the Wednesday call between Trump, Zelenskyy and European leaders.

    “[Trump] took a somewhat stronger line against Putin than was expected, saying the Russian president faced severe consequences if he didn’t meaningfully engage in ceasefire talks,” he noted.

     

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  • Apple’s New Low-Cost iPad Could Come with the A18 Chip and Apple Intelligence, Report Says

    Apple’s New Low-Cost iPad Could Come with the A18 Chip and Apple Intelligence, Report Says

    Apple’s rumored new low-cost iPad could come with two big changes that current devices can’t touch. Rumor has it that Apple’s low-cost iPad will come with an A18 chip, based on code references leaked on Wednesday. MacRumors reported the rumor on Thursday, just weeks ahead of Apple’s rumored Sept. 9 event, where the iPhone 17 is expected to be revealed. 

    Here’s what we’ve gathered so far, but we won’t know all the details until Apple makes its official announcement. 

    The big difference from the current iPad 

    The latest iPad has an A16 processor, which is faster than previous generations but doesn’t support Apple Intelligence, which is Apple’s own AI.

    Now, there’s a rumor that the upcoming iPad could bring the best of both worlds together, if Apple adds the A18 chip to give the new iPad more functionality. 

    What’s so special about the A18 chip?

    ai-atlas-tag.png

    The A16 chip is already a faster processor with better performance compared to previous versions, but it’s not enough for Apple Intelligence. The upgrade could mean even better speed and performance, but the big difference is the capability for Apple Intelligence features that the A16 chip cannot handle. 

    The A18 chip isn’t new to Apple. It’s already in the iPhone 16, iPhone 16 Plus and iPhone 16e. Apple’s upcoming low-cost MacBook is also expected to have the A18 Pro processor, a report says. 

    When will Apple release its new iPad?

    Apple hasn’t confirmed that the new low-cost iPad is coming, but MacRumors pointed out that Apple usually releases iPads in the first half of the year. The current 11th generation iPad was released in March 2025. It’s likely we’ll know more about the cost, features and specs soon. 


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